Apoptosis is programmed cell death, the body's normal method of disposing of damaged, unwanted, or unneeded cells.
AZoLifeSciences speaks to Dr. James Whittle and Professor Geoff Lindeman about their research in enhancing breast cancer therapy through killing sleeper cells.
When our neurons -- the principle cells of the brain -- die, so do we. Most neurons are created during embryonic development and have no "backup" after birth.
The cover for issue 29 of Oncotarget features Figure 5, "In vivo effects of treatment with L-Grb2 in combination with anti-angiogenic therapy in an ovarian tumor model," by Lara, et al. which reported that adaptor proteins such as growth factor receptor-bound protein-2 play important roles in cancer cell signaling.
A new study has revealed that environmental pollutants pose toxicological risks to finless porpoises (Neophocaena asiaeorientalis).
Bacteria of the genus Bartonella are parasites that can be transmitted to humans via insect bites and animal scratches, resulting in an infection known as "bartonellosis." Cat-scratch disease and trench fever are forms of bartonellosis caused by different Bartonella species infecting humans.
Scientists from the Immanuel Kant Baltic Federal University and National University of Science and Technology "MISiS" have studied how magnet nanoparticles affect cancer cells in the human liver.
Silicone molecules extracted from breast implants can trigger the processes in human cells that result in apoptosis.
When pathogenic bacteria infect, the body’s immune system attempts to remove these invaders. One method to do this is to trigger an inflammatory response.
Ludwig-Maximilian-Universitaet in Munich researchers have discovered a hitherto unknown molecular function of a specific microRNA that preserves integrity of the endothelium and reduces the risk of atherosclerosis.
For the first time, researchers from Van Andel Institute have explained the near-atomic level structure of a molecular route that has crucial roles to play in the regulation of blood pressure, cell death, inflammation, and human development.
The p53 gene is crucial in cell biology and thus cell replacement therapy. The function of this gene is to control the cell cycle and stop tumor formation.
Apoptosis is a form of 'cell suicide', in which a series of programmed molecular steps in a cell lead to its death.
Researchers at the University of Bayreuth have discovered a natural protective mechanism that leads to the programmed death of potentially diseased cells.
This story looks into the potential to treat various types of cancers using cells and the process of apoptosis.
A team at the Centro Nacional de Investigaciones Cardiovasculares, working in partnership with researchers at Mount Sinai Hospital in New York, has discovered a new molecular mechanism mediated by nuclear receptors that determines the identity and expansion of macrophages--one of the cell types that act as immune sentinels in the body.
An international group of scientists from the University of Zurich, the University Hospital Zurich, Heidelberg, and Glasgow have discovered a new function of MCL1, the protein that regulates cell death.
According to research conducted in fruit flies at the Francis Crick Institute, the University of Lausanne (UNIL) and the Max Planck Institute for Chemical Ecology, zombie cells tend to develop into functional neurons when the death of neurons is stopped during brain growth.